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Equine facial paralysis

Causes[ edit ] After the conus medullaris, the canal contains a mass of nerves the cauda equina or "horse-tail" that branches off the lower end of the spinal cord and contains the nerve roots from L and S The nerve roots from L4-S4 join in the sacral plexus which affects the sciatic nerve, which travels caudally toward the feet. Compression, trauma or other damage to this region of the spinal canal can result in cauda equina syndrome. The symptoms may also appear as a temporary side-effect of a sacral extra-dural injection:
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Head Shaking May Have a Variety of Causes

Equine facial paralysis
Equine facial paralysis
Equine facial paralysis
Equine facial paralysis
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VIRAL DISEASES | Zoology for IAS, IFoS and other competitive exams

The EIA virus is mechanically transmitted from one horse to another by the bloodsucking horse flies, deer flies Tabanus , stable flies Stomoxys spp. Symptoms include recurrent fever, weight loss, an enlarged spleen, anemia, and swelling of the lower chest, abdominal wall, penile sheath, scrotum, and legs. Horse tires easily due to a recurrent fever and anemia, may relapse to acute form even several years after the original attack. The EIA virus is a slow acting virus of the lenti-retrovirus group.
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Snake bite in southern Africa: diagnosis and management

Allelic disorders with overlapping phenotypes include paramyotonia congenita and the potassium-aggravated myotonias An important clinical difference between the 2 entities is represented by the triggers of attacks of weakness, e. Myotonia was sometimes coexistent.
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Mechanism[ edit ] The toxin is the protein botulinum toxin produced under anaerobic conditions where there is no oxygen by the bacterium Clostridium botulinum. Clostridium botulinum is a large anaerobic Gram-positive bacillus that forms subterminal endospores. The toxin from all of these acts in the same way and produces similar symptoms: Botulinum toxin is broken into 8 neurotoxins labeled as types A, B, C [C1, C2], D, E, F, and G , which are antigenically and serologically distinct but structurally similar. Human botulism is caused mainly by types A, B, E, and rarely F.
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